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These conclusions position EBNA-3a just like a essential modulator of B cellular proliferation and survival during EBV latency, and as a relevant CD8⁺ T mobile purpose in the two basic immunological experiments and translational examine.
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EBNA-3a appears to control a subset of mobile genes, lots of which overlap with People controlled by EBNA2. The two proteins can act antagonistically or cooperatively to control gene expression.
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